GSE136868

syn77077388

Created By Aditya Nath aditya.nath

doi: 10.1038/s41467-020-16786-5
title: NTRK2 splice variant, TrkB.T1, is the predominant isoform in gliomas and enhances gliomagenesis via PI3K signaling
funder: NIH-NCI
series:
creator: Pattwell Siobhan Arora Sonali Cimino Patrick Ozawa Tatsuya Szulzewsky Frank Hoellerbauer Pia Hoffstrom Benjamin Boiani Norman Bolouri Hamid Correnti Colin Silber John Paddison Patrick Holland Eric
license: CC-BY 4.0
species: Homo sapiens
studyId: syn7349757
subject: NSC-U5
ageGroup:
citation: Pattwell SS et al. (2020). A kinase-deficient NTRK2 splice variant predominates in glioma and amplifies several oncogenic signaling pathways. Nat Commun. 11(1):2977. doi: 10.1038/s41467-020-16786-5
dataType: gene expression
keywords: NSC-U5
accessType: Open Access
contributor:
description: Independent scientific achievements have led to the discovery of aberrant splicing patterns in oncogenesis while more recent advances have uncovered novel gene fusions involving NTRK2 in gliomas. The exploration of NTRK2 splice variants in normal and neoplastic brain provides an intersection of these two rapidly evolving fields – alternative splicing and NTRK2 involvement in brain tumors. Tropomyosin receptor B (TrkB), encoded by the NTRK2 gene, is known for critical roles in neuronal surviva...
grantNumber: CA193461
diseaseFocus:
downloadType: Synapse Hosted
alternateName: GSE136868
manifestation:
specimenCount: 9
yearProcessed: 2019
countryOfOrigin:
individualCount: 9
visualizeDataOn:
dataUseModifiers:
conditionsOfAccess: No restriction
measurementTechnique: RNA-seq
externalRepositoryUri: geo:GSE136868
includedInDataCatalog:

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